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• Mutations of single residues in the complexin N-terminus exhibit distinct phenotypes in synaptic vesicle fusion
  Article (Journal), Online Resource
• SNAP25 disease mutations change the energy landscape for synaptic exocytosis due to aberrant SNARE interactions
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• Reduced protein stability of 11 pathogenic missense STXBP1/MUNC18-1 variants and improved disease prediction
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• Arrangements of proteins at reconstituted synaptic vesicle fusion sites depend on membrane separation
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• The interaction of Munc18-1 helix 11 and 12 with the central region of the VAMP2 SNARE motif is essential for SNARE templating and synaptic transmission
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Co-Autoren

• Malsam, Jörg
• Malsam, Andrea
• André, Timon
• Krause, Jean Michel
• Parisotto, Daniel

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