Distribution of the CCR5-delta32 deletion in Southwest Germany

A 32 base pair deletion in the c-c chemokine receptor gene 5 (CCR5) leads to an inactive protein. Carriers of this deletion must have had a selective advantage because the allelic frequency of the CCR5-delat32 mutation is much higher than expected. Furthermore, there is a decline from North to South...

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Hauptverfasser: Hütter, Gero (VerfasserIn) , Blüthgen, Christian (VerfasserIn) , Elvers-Hornung, Susanne (VerfasserIn) , Klüter, Harald (VerfasserIn) , Bugert, Peter (VerfasserIn)
Dokumenttyp: Article (Journal)
Sprache:Englisch
Veröffentlicht: 1. August 2015
In: Journal of biological and clinical anthropology
Year: 2015, Jahrgang: 72, Heft: 3, Pages: 303-309
ISSN:2363-7099
Online-Zugang: Volltext
Verfasserangaben:Gero Hütter, Christian Blüthgen, Susanne Elvers-Hornung, Harald Klüter, Peter Bugert

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520 |a A 32 base pair deletion in the c-c chemokine receptor gene 5 (CCR5) leads to an inactive protein. Carriers of this deletion must have had a selective advantage because the allelic frequency of the CCR5-delat32 mutation is much higher than expected. Furthermore, there is a decline from North to South Europe. For Germany there are just very few cross-sectional surveys available. Here we investigated a large number of healthy blood donors from Northern Baden-Wuerttemberg. We observed an allelic frequency of 9.21 % of the CCR5-delta32 deletion. The distribution did not follow the Hardy-Weinberg equilibrium suggesting that homozygous carriers of the deletion were overrepresented in this random sample. 
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