RAGE does not contribute to renal injury and damage upon ischemia/reperfusion-induced injury

The receptor for advanced glycation end products (RAGE) mediates a variety of inflammatory responses in renal diseases, but its role in renal ischemia/reperfusion (I/R) injury is unknown. We showed that during renal I/R, RAGE ligands HMGB1 and S100B are expressed. However, RAGE deficiency does not a...

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Bibliographische Detailangaben

Hauptverfasser:	Dessing, Mark C. (VerfasserIn) , Nawroth, Peter Paul (VerfasserIn) , Bierhaus, Angelika (VerfasserIn)
Dokumenttyp:	Article (Journal)
Sprache:	Englisch
Veröffentlicht:	2012
In:	Journal of innate immunity Year: 2012, Jahrgang: 4, Heft: 1, Pages: 80-85
ISSN:	1662-8128
DOI:	10.1159/000334251
Online-Zugang:	Verlag, Volltext: http://dx.doi.org/10.1159/000334251 Verlag, Volltext: https://www.karger.com/Article/FullText/334251
Verfasserangaben:	Mark C. Dessing, Wilco P. Pulskens, Gwendoline J. Teske, Loes M. Butter, Tom van der Poll, Huan Yang, Kevin J. Tracey, Peter P. Nawroth, Angelika Bierhaus, Sandrine Florquin, Jaklien C. Leemans