Inhibitory Regulation of Excitatory Neurotransmission
Within the central and peripheral nervous systems of animals, including man, inhibition is crucial to counterbalance excitatory neurotransmission, which is predominantly mediated by glutamate and its receptors. Although, particularly in brain, much of this inhibition is provided by classical post-sy...
Gespeichert in:
| Weitere Verfasser: | |
|---|---|
| Dokumenttyp: | Edited Volume |
| Sprache: | Englisch |
| Veröffentlicht: |
Berlin, Heidelberg
Springer-Verlag Berlin Heidelberg
2008
|
| Schriftenreihe: | Results and Problems in Cell Differentiation
44 SpringerLink Bücher |
| Volumes / Articles: | Show Volumes / Articles. |
| DOI: | 10.1007/978-3-540-72602-9 |
| Schlagworte: | |
| Online-Zugang: | Verlag, Volltext: https://doi.org/10.1007/978-3-540-72602-9 Resolving-System, lizenzpflichtig, Volltext: http://dx.doi.org/10.1007/978-3-540-72602-9 Cover: https://swbplus.bsz-bw.de/bsz273087231cov.jpg |
| Verfasserangaben: | edited by Mark G. Darlison |
MARC
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| 505 | 8 | |a Preliminaries; Preface; Contents; Regulation of Excitation by GABAA Receptor Internalization; Regulation of Excitability by Extrasynaptic GABAA Receptors; GABAC Receptors in Retina and Brain; Presynaptic Ionotropic GABA Receptors; The Role of GABAB Receptors in the Regulation of Excitatory Neurotransmission; GABAergic Control of CA3-driven Network Events in the Developing Hippocampus; Regulation of Excitation by Glycine Receptors; Regulation of Excitability by Potassium Channels; Modulation of Excitation byMetabotropic Glutamate Receptors | |
| 505 | 8 | |a Presynaptic Inhibition of Glutamate Release by Neuropeptides: Use-Dependent Synaptic ModificationRegulation of Excitation by GABA Neurotransmission: Focus on Metabolismand Transport; Human Disorders Caused by the Disruption of the Regulation of Excitatory Neurotransmission; Subject Index; | |
| 520 | |a Within the central and peripheral nervous systems of animals, including man, inhibition is crucial to counterbalance excitatory neurotransmission, which is predominantly mediated by glutamate and its receptors. Although, particularly in brain, much of this inhibition is provided by classical post-synaptic GABAA receptors, many other proteins and mechanisms regulate excitation. These exist both to "fine tune" neurotransmission and to prevent overexcitation that could lead to conditions such as epilepsy and excitotoxicity, which can result in cell death.This book reviews aspects of GAB | ||
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| 650 | 0 | |a Neurobiology | |
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