Vnenčák, M., Paul, M. H., Hick, M., Schwarzacher, S. W., Turco, D. D., Müller, U. C., . . . Jedlicka, P. (2015). Deletion of the amyloid precursor-like protein 1 (APLP1) enhances excitatory synaptic transmission, reduces network inhibition but does not impair synaptic plasticity in the mouse dentate gyrus. The journal of comparative neurology, 523(11), . https://doi.org/10.1002/cne.23766
Chicago-Zitierstil (17. Ausg.)Vnenčák, Matej, Mandy H. Paul, Meike Hick, Stephan W. Schwarzacher, Domenico Del Turco, Ulrike C. Müller, Thomas Deller, und Peter Jedlicka. "Deletion of the Amyloid Precursor-like Protein 1 (APLP1) Enhances Excitatory Synaptic Transmission, Reduces Network Inhibition but Does Not Impair Synaptic Plasticity in the Mouse Dentate Gyrus." The Journal of Comparative Neurology 523, no. 11 (2015). https://doi.org/10.1002/cne.23766.
MLA-Zitierstil (9. Ausg.)Vnenčák, Matej, et al. "Deletion of the Amyloid Precursor-like Protein 1 (APLP1) Enhances Excitatory Synaptic Transmission, Reduces Network Inhibition but Does Not Impair Synaptic Plasticity in the Mouse Dentate Gyrus." The Journal of Comparative Neurology, vol. 523, no. 11, 2015, https://doi.org/10.1002/cne.23766.