Transient neuromotor phenotype in transgenic spastic mice expressing low levels of glycine receptor β‐subunit: an animal model of startle disease
Startle disease or hereditary hyperekplexia has been shown to result from mutations in the α1‐subunit gene of the inhibitory glycine receptor (GlyR). In hyperekplexia patients, neuromotor symptoms ge...
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| Hauptverfasser: | , , , , , |
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| Dokumenttyp: | Article (Journal) |
| Sprache: | Englisch |
| Veröffentlicht: |
2000
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| In: |
European journal of neuroscience
Year: 2000, Jahrgang: 12, Heft: 1, Pages: 27-32 |
| ISSN: | 1460-9568 |
| DOI: | 10.1046/j.1460-9568.2000.00877.x |
| Online-Zugang: | Verlag, lizenzpflichtig, Volltext: https://doi.org/10.1046/j.1460-9568.2000.00877.x Verlag, lizenzpflichtig, Volltext: https://onlinelibrary.wiley.com/doi/10.1046/j.1460-9568.2000.00877.x 
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| Verfasserangaben: | Lore Becker, Bettina Hartenstein, Johannes Schenkel, Jochen Kuhse, Heinrich Betz and Hans Weiher |
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| 520 | |a Startle disease or hereditary hyperekplexia has been shown to result from mutations in the α1‐subunit gene of the inhibitory glycine receptor (GlyR). In hyperekplexia patients, neuromotor symptoms ge... | ||
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