RAGE and ICAM-1 differentially control leukocyte recruitment during acute inflammation in a stimulus-dependent manner
The receptor for advanced glycation endproducts, RAGE, is involved in the pathogenesis of many inflammatory conditions, which is mostly related to its strong activation of NF-κB but also due to its function as ligand for the β2-integrin Mac-1. To further dissect the stimulus-dependent role of RAGE o...
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| Main Authors: | , , , , , , , , , , |
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| Format: | Article (Journal) |
| Language: | English |
| Published: |
4 October 2011
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| In: |
BMC immunology
Year: 2011, Volume: 12, Pages: 1-12 |
| ISSN: | 1471-2172 |
| DOI: | 10.1186/1471-2172-12-56 |
| Online Access: | Verlag, lizenzpflichtig, Volltext: https://doi.org/10.1186/1471-2172-12-56
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| Author Notes: | David Frommhold, Anna Kamphues, Susanne Dannenberg, Kirsten Buschmann, Victoria Zablotskaya, Raphaela Tschada, Baerbel Lange-Sperandio, Peter P. Nawroth, Johannes Poeschl, Angelika Bierhaus and Markus Sperandio |
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| 520 | |a The receptor for advanced glycation endproducts, RAGE, is involved in the pathogenesis of many inflammatory conditions, which is mostly related to its strong activation of NF-κB but also due to its function as ligand for the β2-integrin Mac-1. To further dissect the stimulus-dependent role of RAGE on leukocyte recruitment during inflammation, we investigated β2-integrin-dependent leukocyte adhesion in RAGE-/-and Icam1-/-mice in different cremaster muscle models of inflammation using intravital microscopy. | ||
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