Hagenmüller, M., Malekar, P., Fieger, C., Weiss, C., Buß, S. J., Wolf, D. G., . . . Hardt, S. (2010). Depletion of mammalian target of rapamycin (mTOR) via siRNA mediated knockdown leads to stabilization of β-catenin and elicits distinct features of cardiomyocyte hypertrophy. FEBS letters, 584(1), . https://doi.org/10.1016/j.febslet.2009.10.080
Chicago-Zitierstil (17. Ausg.)Hagenmüller, Marco, Pratima Malekar, Christiane Fieger, Celine Weiss, Sebastian Johannes Buß, David Gregor Wolf, Hugo Katus, und Stefan Hardt. "Depletion of Mammalian Target of Rapamycin (mTOR) via SiRNA Mediated Knockdown Leads to Stabilization of β-catenin and Elicits Distinct Features of Cardiomyocyte Hypertrophy." FEBS Letters 584, no. 1 (2010). https://doi.org/10.1016/j.febslet.2009.10.080.
MLA-Zitierstil (9. Ausg.)Hagenmüller, Marco, et al. "Depletion of Mammalian Target of Rapamycin (mTOR) via SiRNA Mediated Knockdown Leads to Stabilization of β-catenin and Elicits Distinct Features of Cardiomyocyte Hypertrophy." FEBS Letters, vol. 584, no. 1, 2010, https://doi.org/10.1016/j.febslet.2009.10.080.