Recipient Toll-like receptors contribute to chronic graft dysfunction by both MyD88- and TRIF-dependent signaling
Toll-like receptors (TLRs) recognize specific molecular patterns derived from microbial components (exogenous ligands) or stressed cells (endogenous ligands). Stimulation of these receptors leads to a pronounced inflammatory response in a variety of acute animal models. Chronic allograft dysfunction...
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| Hauptverfasser: | , , , , , , , , , , |
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| Dokumenttyp: | Article (Journal) |
| Sprache: | Englisch |
| Veröffentlicht: |
14 January 2010
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| In: |
Disease models & mechanisms
Year: 2010, Jahrgang: 3, Heft: 1/2, Pages: 92-103 |
| ISSN: | 1754-8411 |
| DOI: | 10.1242/dmm.003533 |
| Online-Zugang: | Resolving-System, lizenzpflichtig, Volltext: https://doi.org/10.1242/dmm.003533 Verlag, lizenzpflichtig, Volltext: https://journals.biologists.com/dmm/article/3/1-2/92/2409/Recipient-Toll-like-receptors-contribute-to 
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| Verfasserangaben: | Shijun Wang, Christoph Schmaderer, Eva Kiss, Claudia Schmidt, Mahnaz Bonrouhi, Stefan Porubsky, Norbert Gretz, Liliana Schaefer, Carsten J. Kirschning, Zoran V. Popovic and Hermann-Josef Gröne |
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| 520 | |a Toll-like receptors (TLRs) recognize specific molecular patterns derived from microbial components (exogenous ligands) or stressed cells (endogenous ligands). Stimulation of these receptors leads to a pronounced inflammatory response in a variety of acute animal models. Chronic allograft dysfunction (CAD) was regarded as a candidate disease to test whether TLRs influence chronic fibrosing inflammation.... | ||
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