Li, S., Li, H., Bennewitz, K., Poschet, G., Büttner, M., Haußer-Siller, I., . . . Kroll, J. (2025). Combined loss of glyoxalase 1 and aldehyde dehydrogenase 3a1 amplifies dicarbonyl stress, impairs proteasome activity resulting in hyperglycemia and activated retinal angiogenesis. Metabolism, 165, . https://doi.org/10.1016/j.metabol.2025.156149
Chicago Style (17th ed.) CitationLi, Shu, Hao Li, Katrin Bennewitz, Gernot Poschet, Michael Büttner, Ingrid Haußer-Siller, Julia Szendrödi, Peter Paul Nawroth, and Jens Kroll. "Combined Loss of Glyoxalase 1 and Aldehyde Dehydrogenase 3a1 Amplifies Dicarbonyl Stress, Impairs Proteasome Activity Resulting in Hyperglycemia and Activated Retinal Angiogenesis." Metabolism 165 (2025). https://doi.org/10.1016/j.metabol.2025.156149.
MLA (9th ed.) CitationLi, Shu, et al. "Combined Loss of Glyoxalase 1 and Aldehyde Dehydrogenase 3a1 Amplifies Dicarbonyl Stress, Impairs Proteasome Activity Resulting in Hyperglycemia and Activated Retinal Angiogenesis." Metabolism, vol. 165, 2025, https://doi.org/10.1016/j.metabol.2025.156149.