The thrombopoietin receptor P106L mutation functionally separates receptor signaling activity from thrombopoietin homeostasis

Key Points. The c-Mpl activity in downstream signaling and in platelet homeostasis can be functionally separated.The c-Mpl platelet homeostasis depends on corr

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Bibliographic Details
Main Authors:	Stockklausner, Clemens (Author) , Klotter, Anne-Christine (Author) , Dickemann, Nicole (Author) , Kuhlee, Isabelle Nadine (Author) , Duffert, Christin (Author) , Kerber, Carolin (Author) , Gehring, Niels H. (Author) , Kulozik, Andreas (Author)
Format:	Article (Journal)
Language:	English
Published:	2015
In:	Blood Year: 2014, Volume: 125, Issue: 7, Pages: 1159-1169
ISSN:	1528-0020
DOI:	10.1182/blood-2014-07-587170
Online Access:	Verlag, lizenzpflichtig, Volltext: https://doi.org/10.1182/blood-2014-07-587170 Verlag, lizenzpflichtig, Volltext: https://ashpublications.org/blood/article/125/7/1159/34190/The-thrombopoietin-receptor-P106L-mutation
Author Notes:	Clemens Stockklausner, Anne-Christine Klotter, Nicole Dickemann, Isabelle N. Kuhlee, Christin M. Duffert, Carolin Kerber, Niels H. Gehring, and Andreas E. Kulozik

Description
Summary:	Key Points. The c-Mpl activity in downstream signaling and in platelet homeostasis can be functionally separated.The c-Mpl platelet homeostasis depends on corr
Item Description:	Prepublished online as Blood First Edition paper, December 23, 2014 Gesehen am 23.06.2020
Physical Description:	Online Resource
ISSN:	1528-0020
DOI:	10.1182/blood-2014-07-587170