APA (7th ed.) Citation

Stockklausner, C., Klotter, A., Dickemann, N., Kuhlee, I. N., Duffert, C., Kerber, C., . . . Kulozik, A. (2015). The thrombopoietin receptor P106L mutation functionally separates receptor signaling activity from thrombopoietin homeostasis. Blood, 125(7), . https://doi.org/10.1182/blood-2014-07-587170

Chicago Style (17th ed.) Citation

Stockklausner, Clemens, Anne-Christine Klotter, Nicole Dickemann, Isabelle Nadine Kuhlee, Christin Duffert, Carolin Kerber, Niels H. Gehring, and Andreas Kulozik. "The Thrombopoietin Receptor P106L Mutation Functionally Separates Receptor Signaling Activity from Thrombopoietin Homeostasis." Blood 125, no. 7 (2015). https://doi.org/10.1182/blood-2014-07-587170.

MLA (9th ed.) Citation

Stockklausner, Clemens, et al. "The Thrombopoietin Receptor P106L Mutation Functionally Separates Receptor Signaling Activity from Thrombopoietin Homeostasis." Blood, vol. 125, no. 7, 2015, https://doi.org/10.1182/blood-2014-07-587170.