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Glutathione is a factor of resistance of Jurkat leukemia cells to nitric oxide‐mediated apoptosis

We have previously reported that nitric oxide (NO) stimulates apoptosis in different human neoplastic lymphoid cell lines through mitochondrial damage (including degradation of cardiolipin, a major mitochondrial lipid) followed by activation of caspases.

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Bibliographic Details
Main Authors: Umansky, Viktor (Author) , Dröge, Wulf (Author)
Format: Article (Journal)
Language:English
Published: 12 June 2000
In: Journal of cellular biochemistry
Year: 2000, Volume: 78, Issue: 4, Pages: 578-587
ISSN:1097-4644
DOI:10.1002/1097-4644(20000915)78:4<578::AID-JCB7>3.0.CO;2-A
Online Access:Verlag, lizenzpflichtig, Volltext: https://doi.org/10.1002/1097-4644(20000915)78:4<578::AID-JCB7>3.0.CO;2-A
Verlag, lizenzpflichtig, Volltext: https://onlinelibrary.wiley.com/doi/10.1002/1097-4644(20000915)78:4<578::AID-JCB7>3.0.CO;2-A
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Author Notes:Victor Umansky, Marian Rocha, Raoul Breitkreutz, Steffen Hehner, Mariana Bucur, Natalie Erbe, Wulf Dröge, and Alexey Ushmorov
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Summary:We have previously reported that nitric oxide (NO) stimulates apoptosis in different human neoplastic lymphoid cell lines through mitochondrial damage (including degradation of cardiolipin, a major mitochondrial lipid) followed by activation of caspases.
Item Description:Gesehen am 03.02.2021
Physical Description:Online Resource
ISSN:1097-4644
DOI:10.1002/1097-4644(20000915)78:4<578::AID-JCB7>3.0.CO;2-A

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