Immune (dys)regulation in multiple sclerosis: role of the CD95-CD95 ligand system

T cells specific for myelin antigens are thought to initiate and perpetuate the immune process behind multiple sclerosis. Apoptosis is a key regulatory mechanism that determines T-cell fate and might mediate tissue damage. Here, Frauke Zipp, Peter Krammer and Michael Weller discuss the role of the C...

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Bibliographic Details
Main Authors: Zipp, Frauke (Author) , Krammer, Peter H. (Author) , Weller, Michael (Author)
Format: Article (Journal)
Language:English
Published: 24 November 1999
In: Immunology today
Year: 1999, Volume: 20, Issue: 12, Pages: 550-554
ISSN:1355-8242
DOI:10.1016/S0167-5699(99)01545-5
Online Access:Verlag, lizenzpflichtig, Volltext: https://doi.org/10.1016/S0167-5699(99)01545-5
Verlag, lizenzpflichtig, Volltext: https://www.sciencedirect.com/science/article/pii/S0167569999015455
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Author Notes:Frauke Zipp, Peter H. Krammer and Michael Weller
Description
Summary:T cells specific for myelin antigens are thought to initiate and perpetuate the immune process behind multiple sclerosis. Apoptosis is a key regulatory mechanism that determines T-cell fate and might mediate tissue damage. Here, Frauke Zipp, Peter Krammer and Michael Weller discuss the role of the CD95-CD95 ligand system in multiple sclerosis.
Item Description:Gesehen am 03.03.2021
Physical Description:Online Resource
ISSN:1355-8242
DOI:10.1016/S0167-5699(99)01545-5