Amino acid metabolism in liver disease
The impairment of transsulphuration during methionine degradation in hepatic failure can be counteracted by treatment with S-adenosylmethionine. Regarding the pathogenesis of hepatic encephalopathy, no convincing evidence exists for tryptophan, glutamine or glutamate being involved. Portal-systemic...
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| Main Authors: | , , |
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| Format: | Article (Journal) |
| Language: | English |
| Published: |
1999
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| In: |
Current opinion in clinical nutrition & metabolic care
Year: 1999, Volume: 2, Issue: 1, Pages: 47-53 |
| ISSN: | 1473-6519 |
| Online Access: | Verlag, lizenzpflichtig, Volltext: https://journals.lww.com/co-clinicalnutrition/Fulltext/1999/01000/Amino_acid_metabolism_in_liver_disease.9.aspx
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| Author Notes: | Eggert Holm, Oliver Sedlaczek, Eva Grips |
| Summary: | The impairment of transsulphuration during methionine degradation in hepatic failure can be counteracted by treatment with S-adenosylmethionine. Regarding the pathogenesis of hepatic encephalopathy, no convincing evidence exists for tryptophan, glutamine or glutamate being involved. Portal-systemic shunting-induced hyperammonaemia may reduce plasma branched-chain amino acids. The glucose effect on urea synthesis does not exist in cirrhosis. |
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| Item Description: | Gesehen am 23.03.2022 |
| Physical Description: | Online Resource |
| ISSN: | 1473-6519 |